Case Of Constrictive Pericarditis Research Articles

Large Organized Intrapericardial Hematoma Presenting as a Case of Constrictive Pericarditis and Superior Vena Cava Syndrome

Superior vena cava syndrome and constrictive pericarditis are rare complications of intrapericardial hematoma. We herein report a young patient presenting with a mass attached to the heart causing pressure on the right atrium and superior vena cava, and leading to superior vena cava syndrome and constrictive pericarditis. The exact pathogenesis was not determined but possibly was related to pericardiocentesis 29 years prior to presentation. Diagnosis was established at surgery and confirmed by histopathology. Resolution was partial because complete resection was not possible due to adherence of the mass to the right ventricular wall.

冠動脈バイパス術後2カ月目から右心不全症状を呈したが，心膜の肥厚・石灰化を伴わず収縮性心膜炎として加療するまでに時間を要した1例

冠動脈バイパス術後2カ月目から右心不全症状を呈したが，心膜の肥厚・石灰化を伴わず収縮性心膜炎として加療するまでに時間を要した1例

A case of constrictive pericarditis in a patient with rheumatoid arthritis

Система комплексной поддержки и сопровождения научного журнала Elpub позволяет запустить двуязычный сайт журнала со встроенной системой электронной редакции в соответствии с лучшими издательскими практиками, а так же предусматривает техническую и методическую поддержку со стороны специалистов НЭИКОН.

A case of constrictive pericarditis caused by adhesion of the benign mediastinal cyst

A case of constrictive pericarditis caused by adhesion of the benign mediastinal cyst

Constrictive pericarditis in a renal transplant recipient with tuberculosis

Tuberculosis is a common cause of pericarditis in the developing countries and constrictive pericarditis is a serious sequel. There are only three cases of constrictive pericarditis in kidney transplant recipients previously reported in literature. Here, we report a case of constrictive pericarditis developing in a renal transplant recipient while on antituberculous therapy for tuberculous pleural effusion.

Three-Dimensional Transesophageal Echocardiography: Diagnosing the Extent of Pericarditis Constrictiva and Intraoperative Surgical Support

The traditional two-dimensional transthoracic echocardiography (2DTTE) has limitations in demonstrating the extent of pericardial thickening in constrictive pericarditis (CP) because of poor transmission of ultrasound through the thickened anterior pericardial structures. We describe a case of CP, of unknown etiology, in which transesophageal 3DTEE equalled the accuracy of cardiac magnetic resonance in demonstrating the extent of pericardial thickening in CP.

Authors’ Names in a GlobalizedAmerican Journal of Neuroradiology

Constrictive pericarditis is present when a fibrotic, thickened, and adherent pericardium restricts diastolic filling of the heart. Several drugs can cause pericarditis, which can lead to chronic pericardial constriction. A case of constrictive pericarditis in a patient receiving the antiparkinsonian drug pergolide is reported.

Congestive Hepatopathy in the Setting of Undiagnosed Constrictive Pericarditis

Purpose: Background: Ascites is a common finding in patients with cirrhosis. The non-hepatic causes of ascites include venous or lymphatic obstruction, nephrotic syndrome, and congestive heart failure. Constrictive pericarditis (CP) is characterized by pericardial inflammation and subsequent scarring and contracture of the pericardium. The clinical manifestation of CP is usually congestive heart failure (CHF) which results in a “nutmeg liver” due to chronic congestive changes in the liver. Histologic findings in the liver include sinusoidal dilatation, fibrosis around spared portal regions and hemorrhagic necrosis in zone 3 of the hepatic lobule. Recently, the prevalence of CP has declined significantly due to increased focus on preventive medicine and improved diagnostic capability. Thus, CP with congestive hepatopathy (CH) has not been reported. We report a case of CH with ascites caused by CP. Case Report: A 78 year-old male with history of hypertension, diabetes, and dyslipidemia presented with gradual increase in abdominal girth over 5–8 months; this was attributed to ascites. The patient denied dyspnea at rest or on exertion and orthopnea. He denied alcohol or drug use, and history of previous liver disease or radiation exposure. Patient's mild abdominal pain was relieved by a paracentesis; 5 liters of transudative fluid were removed. The serum ascites albumin gradient was 1.0. An abdominal CT scan showed cirrhosis and extensive calcification in the inferior pericardium. The latter finding was confirmed on a CT scan of the chest. Tuberculin skin test was negative. Platelet count was 270,000/μL, sodium 137 mEq/L, creatinine 1.5 mg/dL, prothrombin time 22 seconds, aspartate aminotransferase 19 U/L, alanine aminotransferase 24 U/L, alkaline phosphatase 89 U/L, bilirubin 6.4 mg/dL. Hepatic sinusoidal pressure was 16 mmHg; liver histology was consistent with CH. Transthoracic Echocardiogram revealed biatrial enlargement and normal left ventricular ejection fraction. Right and left heart catheterizations indicated relative equalization of pressures in the cardiac chambers, restrictive physiology and a pulmonary artery wedge pressure of 62 mmHg. The patient underwent an anterior pericardiectomy; however, ascites reaccumulated requiring numerous paracenteses. Discussion: This case illustrates that CP needs to be considered as an etiology of CH, cirrhosis and ascites. In our patient, pericardiectomy was unsuccessful in completely resolving ascites, highlighting the importance of early pericardiectomy and appropriate CHF management to prevent cirrhosis. Although CHF has been associated with CH, this is the first case of CP and CH. Methods: NA Results: NA Conclusion: NA

A case of constrictive pericarditis during cabergoline treatment for hyperprolactinaemia.

Treatment with dopamine agonists has been associated with cardiopulmonary fibrotic reactions, predominantly in patients treated for Parkinson's disease. To our knowledge, these reactions have previously not been associated with low-dose cabergoline treatment for hyperprolactinaemia. A case of constrictive pericarditis in a patient treated with cabergoline for hyperprolactinaemia is presented. The patient has been treated at a county hospital and a university hospital in southern Sweden. A 20-year-old woman with a 3-year history of amenorrhoea was referred to the department in 1992. From 2001 to 2005, she was given cabergoline, 0.5-1.5 mg/week. In 2005 a pericardectomy was performed due to fibrotic, constrictive pericarditis. Our present case suggests that constrictive pericarditis may develop even on low-dose cabergoline, which might indicate that this reaction, as opposed to valvular fibrosis, is not mediated by a 5-HT(2B) agonistic mechanism.

A case of constrictive pericarditis of over ten years' duration treated by pericardiectomy.

A case of constrictive pericarditis of over ten years' duration treated by pericardiectomy.

Pleuropéricardite sévère associée à un traitement par bromocriptine au long cours, revue de la littérature à propos d’un cas

Introduction. – Bromocriptin, member of the class of ergolines, is commonly prescribed as treatment of Parkinson’s disease. Apart from vascular, digestif, neurologic and psychic disorders, the authors report cases of retroperitoneal fibrosis and pleural effusion, as adverse reactions related to the bromocriptin. Synthesis. – About 40 cases of skin, pleural, lung and retroperitoneal attacks were described after long term and high doses of bromocriptin. More ten years ago, the first case of constrictive pericarditis was cited in the medical literature, and the bromocriptin was incriminated as responsible. Since then, two other cases were cited. Our observation is a constrictive pericarditis, found in a 72 years old patient treated with bromocriptin for Parkinson’s disease since five years (cumulative dose intake 73 grams). Investigations aimed to establish etiology were negative. Bromocriptin is suspected and the treatment is discontinued. As in the three other cases, cardiac and neurologic conditions markedly improved after bromocriptin’s withdrawal. A pericardic thickening persists at the echography. Conclusion. – The responsibility of bromocriptin in the etiology of constrictive pericarditis is seldom discussed, because it remains an exclusion diagnosis. Periodic chest X-ray and echocardiography should be considered in patients with long-term bromocriptin treatment.

A case of constrictive pericarditis most likely due to psittacosis

A case of constrictive pericarditis most likely due to psittacosis

A Case of Constrictive Pericarditis with Localized Pericardial Effusion Simulating a Cystic Mass

A Case of Constrictive Pericarditis with Localized Pericardial Effusion Simulating a Cystic Mass

Complications of automatic implantable cardioverter defibrillators: radiographic, CT, and echocardiographic evaluation.

Automatic implantable cardioverter defibrillators (AICDs) were studied in three groups: (a) Serial radiographs were reviewed in 51 clinic patients. Twenty of 96 (21%) AICD patches distorted with time. (b) Thirty-six postoperative computed tomographic (CT) scans of asymptomatic patients revealed that pericardial fluid collections were frequent during the month after surgery but rare beyond that. Echocardiography was insensitive for these collections. CT also demonstrated dense fibrosis around some distorted patches, months after surgery. (c) Five other patients with pericardial infection had distorted patches, and the four studied with CT had fluid beneath their patches. (d) A case of constrictive pericarditis had distorted patches but was not diagnosed with CT. The authors conclude that distorted patches may indicate postoperative complications and that CT is the imaging modality of choice.

Constrictive Pericarditis Due to Infection with Nocardia Asteroides

A case of constrictive pericarditis caused by Nocardia asteroides is reported. Pericardiectomy, combined with proper chemotherapy, gave excellent results in the treatment of the patient herein reported. Even though this case represents a primary infection by Nocardia asteroides, this fungus is known for its opportunistic characteristic, which frequently complicates debilitating diseases of variable etiology.

Constrictive pericarditis following radiation therapy

A case of constrictive pericarditis following radiotherapy for carcinoma of the esophagus is described. An episode of acute pericarditis was observed nine months after completion of therapy, followed shortly by a pericardial effusion and subsequently by signs and symptoms of constrictive pericarditis. A successful pericardiectomy was performed fourteen months after radiation, and to date the patient has shown no recurrence of the malignancy or of congestive heart failure. For most patients treated with conventional doses of radiation to thoracic structures the risk of pericardial disease is probably slight, and continuation of such treatment in the face of minimal changes seems warranted. Patients receiving prophylactic radiotherapy for lymphoma or seminoma and those given radiation over internal mammary nodes or locally for carcinoma of the left breast require more careful consideration as to the need for such therapy, and the dose and method of administration.

Protein-losing gastroenteropathy and cardiac diseases

1) Nine cases (21 per cent) of 43 cases of constrictive perecarditis showed severe hypoproteinemia under 5.0 Gm. per 100 ml., while only one case with such hypoproteinemia was encountered among congestive failure of the other cardiac diseases with high venous pressure over 150mm. saline. This special case was a postoperative teralogy of Fallot accidentally associated with tricuspid and pulmonary insufficiency. 2) Isotope studies on cases with hypoproteinemia under 5.0 Gm. per 100ml. of serum protein (5 cases of idiopathic hypoproteinemia and 3 cases of cardiac diseases) showed definite evidence of protein loss into the gastrointestinal tract. 3) Comparing the venous pressure and duration of signs of venous congestion, among group with hypoproteinemia under 5.0 Gm. per 100 ml., the other group of constrictive pericarditis, and cases with congestive failure associated with tricuspid insufficiency, the important role of hemodynamic abnormalities on severe hypoproteinemia was confirmed. At the same time, it was pointed out that venous congestion is not a sufficient factor but one of necessary factors to explain the hypoproteinemia in individual cases. 4) Preliminary experimental study designed to clarify causal relationship of venous congestion to abnormality of abdominal lymphatic system and protein loss into the gut was reported. 5) A case of constrictive pericarditis whose clinical findings showed some special features inherent to those of idiopathic hypoproteinemia due to protein losing enteropathy, was described, and some clnical statistics concerning some possible factors other than hemodynamic abnormalities were presented.

PROTEIN-LOSING GASTROENTEROPATHY AS CAUSE OF HYPOPROTEINEMIA IN CONSTRICTIVE PERICARDITIS.

A case of constrictive pericarditis with hypoproteinemia was reported with the demonstration of gastrointestinal protein loss and lymphangiectasia of the intestinal mucosa. From these facts hypoproteinemia in cardiac diseases was revealed to be due to gastrointestinal protein loss caused by a disorder of the lymphatic vessels of the small bowel.

Histoplasmosis with massive pericardial effusion and systemic involvement. Report of a case.

IN INCBEASING NUMBEB of cases of histoplasmosis have been reported since Hansmann, Schenken, and DeMonbreun culturedHistoplasma capsulatumfrom cases in 1934. The course, manifestations, organ involvement, epidemiological aspects, endemic, and worldwide distribution of this protean disease are well documented. Heart involvement has been reported with increasing frequency. The etiological agent affects all 3 layers: endocardium, myocardium, and the pericardium. The cases of pericarditis were diagnosed mainly on a sensitivity basis and were manifested as chronic, healed, calcific pericarditis. One case of constrictive pericarditis was added to this category. In addition, a case of an acute form of pericarditis with massive effusion due to histoplasmosis was recently reported. The authors were unable to substantiate any other cases in the literature with special localization in and involvement of the pericardium. The case being reported here coincided with the time their case was published, and we believe that the following may be of

Pericarditis due to histoplasmosis

Two additional cases of pericarditis due to histoplasmosis, including the second known case of constrictive pericarditis, have been reported. This makes a total in the available literature of 3 proved cases, and 6 cases which are presumptive because of positive histoplasmin skin and/or serologic tests in patients with anergy to tuberculin.