Case Of Childhood Research Articles

Lead Poisoning From a Toy Necklace: In Reply

In Reply .— We thank Dr Merritt and his colleagues for their original publications about a case of childhood lead poisoning associated with the ingestion of a lead-contaminated toy necklace1,2 and Dr Merritt for the letter above. He disagrees with our assessment that this child had chronic lead exposure rather than simply 1 “acute” exposure from the swallowed medallion and offer the fact that a thorough environmental investigation failed to uncover a source of lead exposure as evidence that this child's problem was caused exclusively by this ingestion. They also point in their letter to their ongoing neurocognitive surveillance of this youngster rather than offer input on our suggestion that this …

Translocation (11;13)(q23;q14) as the sole abnormality in a childhood de novo acute myelocytic leukemia

We report a case of childhood de novo acute myelocytic leukemia (AML) with hyperleukocytosis with monoblastic features and deranged hemostasic function. G-band karyotyping demonstrated a previously unreported t(11;13)(q23;q14) in metaphase preparations from a fluorodeoxyuridine synchronized 1-day culture of leukophoresed cells. Multicolor fluorescence in situ hybridization revealed no cryptic rearrangements except for the translocation. Reverse transcriptase polymerase chain reaction showed no concomitant positivity of AML1/ETO, BCR/ABL, PML/RARA, and CBFβ/MYH11 resulting from t(8;21)(q22;q22), t(9;22)(q34;q11), t(15;17)(q22;q11), and inv(16) (p13q22), respectively. This report of childhood de novo AML harboring t(11;13)(q23;q14) as the sole cytogenetic abnormality provides more data on the leukemogenesis of de novo AML with a 11q23 rearrangement.

The high cost of improper removal of lead-based paint from housing: a case report.

The costs of lead-based paint hazard control in housing are well documented, but the costs of cleanup after improper, inherently dangerous, methods of removing lead-based paint are not. In this article we report a case of childhood lead poisoning and document the costs of decontamination after uncontained power sanding was used to remove paint down to bare wood from approximately 3,000 ft(2) of exterior siding on a large, well-maintained 75-year-old house in a middle-income neighborhood. After the uncontrolled removal of lead-based paint, interior dust lead levels ranged from 390 to 27,600 micro g Pb/ft(2) (on floors and windowsills) and bare soil lead levels ranged from 360 ppm in the yard to 3,900 ppm along the foundation to 130,000 ppm in the child's play area, well above applicable U.S. Department of Housing and Urban Development/U.S. Environmental Protection Agency standards. The hard costs of decontamination were over $195,000, which greatly exceeds the incremental cost of incorporating lead-safe work practices into repainting. This case report highlights the need to incorporate lead-safe work practices into routine repainting, remodeling, and other renovation and maintenance jobs that may disturb lead-based paint.

Childhood cutaneous mucinosis

A case of childhood cutaneous mucinosis is described. This is a clinical condition that is rarely seen and has only recently been included in the group of primary mucinosis.

Allergic bronchopulmonary cladosporiosis in a child

Since the first descript ion of allergic bronchopu lmona ry aspergillosis (ABPA) in 1952, m a n y repor ts in the l i terature have increased the knowledge about immunolog ic mechanisms, clinical presentat ion, diagnostic criteria, and t r ea tmen t of the disease. 1-3 Aspergillus fumigatus is the mos t comm o n fungus involved, but o ther Aspergillus species and o the r fungi have been r epor t ed as causat ive agents. 4-a2 Candida albicans, 4, 11 Mucor spp., 12 Helminthosporium spp., 5 Stemphylium spp., 6 Torulopsis spp., 7 Curvularia lunata, 8, 22 Rhizopus spp.,l°, 11 Penicillium spp., n Drechslera spp., 8 and Pseudallescher/a spp? 1 are fungi involved in allergic b ronchopul mona ry mycosis ( A B P M ) (Table I). Diagnostic criteria for A B P A are well established in adult patients, 1 but there are few reports of this disease in children. 2,3,9, 22,13 We present a case of childhood A B P M caused by C. herbarum. To our knowledge, this is the only published case of childhood A B P M caused by this fungus. Remiss ion was achieved by adequa te cor t icosteroid t rea tment .

A peculiar case of acquired aphasia with epilepsy in childhood

A case of childhood acquired aphasia with epilepsy (AAE) is reported because of unusual epileptologic and psycholinguistic features. Epileptic seizures were extraordinarily polymorphous, including especially at 4 years 8 months an episode of epilepsia partialis continua. The most striking feature of EEG data was absence of notable activating effect of sleep. Comprehension was never completely lost and seemed partially related to an auditory—verbal unawareness or neglect. Expression deficit was principally a difficulty in naming tasks, and above all prolonged latencies filled with a song-like prolonged vocalization during this task and more rarely in spontaneous speech. There was an anosognosia of these vocalizations. Beside difficulties in naming tasks, major expression disorders were constantly of phonological nature. Word repetition disorder was significantly more severe than both expression and comprehension defects (picture naming and pointing tests). Cases of AAE reported in the literature are heterogeneous, and there is currently no satisfactory explanation of association of an acquired aphasia with epileptic disorders, and no established correlation between a certain type of psycholinguistic defect and a definite clinical or at least electrophysiological epileptic disorder. An effort to define more precisely both psycholinguistic and electro-clinical epileptic disorders might lead to a much needed nosologic classification of AAE.

Neuromaturation and behavior development: The case of childhood lead poisoning.

Neuromaturation and behavior development: The case of childhood lead poisoning.

下がく骨と下たい骨に特徴的変化を呈した小児急性リンパ性白血病の症例

We describe a unique case of 12-years-old girl who was referred to this clinic because of gingival hypertrophy, loosened teeth, and painful leg.A panoramic dental radiograph of the patient revealed a number of abnormal features including cancellous bone destruction with diffuse cloudiness especially noticeable in the premolar and molar regions, loss of alveolar crestal bone, disappearance or thinning of lamina dura of the premolar and molars, disappearance of the mandibular nerve canals, thickening of periodontal membrane space, and enlargement of the both mental foramina.After examinations of the peripheral blood and the bone marrow, the case was diagnosed to be B-cell acute lymphocytic leukemia.Although antileukemic chemotherapy was undertaken at the pediatrics, she died on the 66th hospital day because of interstitial pneumonia.Thus, we suggest that panoramic radiography of the jaws could provide an important clue for diagnosis of some kind of leukemia.

Neuromaturation and behavior development: The case of childhood lead poisoning.

Neuromaturation and behavior development: The case of childhood lead poisoning.

Plutarch's Alexander the Great as Focus for Greek Identity

Plutarch's Alexander the Great as Focus for Greek Identity. The Case of Alexander's Childhood and Youth in Vira Alexandri
 The present paper concentrates on the character of Plutarch’s Alexander and his idealized Greek traits as visible in one particular set of Plutarch’s stories: the narratives on the childhood and youth of Alexander, presented in Vita Alexandri. By presenting him as a Greek hero, with a number of typically heroic and typically Hellenic features, Plutarch transforms the image of the Macedonian king, creating a model for his audience to identify as an embodiment of Greek greatness. While the portrait of Alexander in Plutarch’s Life of Alexander is rather nuanced and not entirely positive (see, for example, his behaviour in the East), Plutarch seems, in the stories of Alexander’s childhood, to be carefully presenting him as a perfect Greek model of a hero and a future leader.

III, 7. A CASE OF SCHIZOPHRENIA IN EARLY CHILDHOOD: (WITH EXAMPLES OF DRAWINGS)

Acta Psychiatrica ScandinavicaVolume 22, Issue S47 p. 323-339 III, 7. A CASE OF SCHIZOPHRENIA IN EARLY CHILDHOOD: (WITH EXAMPLES OF DRAWINGS) NIC WAAL, NIC WAAL Oslo, Norway.Search for more papers by this author NIC WAAL, NIC WAAL Oslo, Norway.Search for more papers by this author First published: December 1947 https://doi.org/10.1111/j.1600-0447.1947.tb03980.xAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat Volume22, IssueS47December 1947Pages 323-339 RelatedInformation