Baboons Research Articles

Evolution of xenoantibodies, after their depletion, in a non‐immunosuppressed baboon

Evolution of xenoantibodies, after their depletion, in a non‐immunosuppressed baboon

The myth of the typical baboon

Evolutionary Anthropology: Issues, News, and ReviewsVolume 13, Issue 4 p. 121-121 News The myth of the typical baboon Erik Willems, Erik Willems [email protected] Evolutionary Anthropology Research Group, Department of Anthropology University of Durham, U.KSearch for more papers by this author Erik Willems, Erik Willems [email protected] Evolutionary Anthropology Research Group, Department of Anthropology University of Durham, U.KSearch for more papers by this author First published: 29 July 2004 https://doi.org/10.1002/evan.20019AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article. Volume13, Issue4September 2004Pages 121-121 RelatedInformation

Baboon dads defend offspring in fights

Baboon dads defend offspring in fights

<Note> Competition between baboons and chimpanzees at Mahale

<Note> Competition between baboons and chimpanzees at Mahale

Absence of PERV specific humoral immune response in baboons after transplantation of porcine cells or organs.

Xenotransplantation of pig organs seems a promising way of overcoming the prevailing limitation on allotransplantation due to donor numbers. However, as porcine endogenous retroviruses (PERVs) can infect human cells in vitro, there is substantial concern regarding the risk of a PERV infection in xenogeneic transplant recipients. Cultured porcine endothelial cells, stimulated peripheral blood mononuclear cells, and pancreatic islet cells can release PERV infectious for human cells in vitro, but it is currently unknown whether PERV is released in vivo, whether these viral particles can infect the transplant recipient, and whether they are pathogenic. In a retrospective study 15 immunosuppressed baboons were tested for a specific immune response against PERV after transplantation of porcine endothelial cells, mononuclear blood cells, and lungs. Anti-PERV antibody expression was analyzed with peptide-based, enzyme-linked immunosorbent assays and highly sensitive Western Blot assays. This xenotransplantation study using nonhuman primates found no evidence of PERV specific humoral immune response. Our data suggest that no productive PERV infection and no continuous PERV release takes place in the nonhuman primates analyzed in this study.

Cobb's Review of Smut's Sex and Friendship in Baboons

Cobb's Review of Smut's Sex and Friendship in Baboons

Preimplantation genetics: an explanation for poor human fertility?

Compared with other mammals, humans as a species are relatively infertile. Various studies suggest that in young, unselected couples, who are trying to conceive, 20–25% of them should be successful each monthly cycle (Bonde et al . 1998; Edwards & Brody, 1995). This compares with an average of 70% in captive baboons for example (Stevens, 1997). Interestingly, the implantation rate after in vitro fertilisation (IVF) at best averages around 20% per embryo transferred (Edwards & Beard, 1999). Evidence is steadily accumulating to prove that the major cause of implantation failure in humans after both in vivo and in vitro fertilisation is the high incidence of chromosomal abnormality. Combining data from cytogenetic studies of spontaneous abortions with those obtained from preimplantation embryos suggests that meiotically derived aneuploidy occurs in 25% of conceptions, an order of magnitude higher than is found in other well studied species such as the mouse (Hassold & Jacobs, 1984; Jamieson, 1994). Additionally, interphase fluorescent in situ hybridisation (FISH) analysis of three day old human embryos has shown that up to 50% are chromosomally mosaic (Delhanty et al . 1997; Munne et al . 1998), further increasing the chance of implantation failure. This review covers the evidence accumulating from a variety of sources that poor fertility in humans has a chromosomal basis.

Bum deal for baboons

Bum deal for baboons

Foraging for Survival: Yearling Baboons in Africa.:Foraging for Survival: Yearling Baboons in Africa.

Foraging for Survival: Yearling Baboons in Africa. Stuart A. Altmann. Chicago: The University of Chicago Press, 1998. 609 pp.

Foraging for Survival. Yearling Baboons in Africa

Foraging for Survival. Yearling Baboons in Africa

Sex and Friendship in Baboons. Barbara B. Smuts

Previous articleNext article No AccessNew Biological BooksSex and Friendship in Baboons. Barbara B. Smuts Vicki K. Bentley-ConditVicki K. Bentley-Condit Search for more articles by this author PDFPDF PLUS Add to favoritesDownload CitationTrack CitationsPermissionsReprints Share onFacebookTwitterLinkedInRedditEmail SectionsMoreDetailsFiguresReferencesCited by The Quarterly Review of Biology Volume 75, Number 4Dec., 2000 Published in association with Stony Brook University Article DOIhttps://doi.org/10.1086/393731 Copyright 2000 The University of ChicagoPDF download Crossref reports no articles citing this article.

A Quantitative Trait Locus for Sodium-Lithium Countertransport Is Linked to Chromosome Four in Baboons

P134 Alterations in cell sodium transport characteristics, such as red blood cell intracellular sodium (ICNa) levels and sodium-lithium countertransport (SLC), are secondary biochemical indices of a primary cell membrane abnormality that have been associated with some forms of essential hypertension. We are studying the effects of genes and environmental factors on hypertension and blood pressure-related phenotypes such as ICNa and SLC in baboons, a non-human primate model for blood pressure regulation. In a previous study of 84 baboons, systolic blood pressure increased with increasing ICNa (p = 0.002) and SLC (p = 0.06). In the current study, genotypes for 280 microsatellite loci, as well as SLC and ICNa data, were determined for 623 baboons comprising 11 pedigrees. The heritabilities of ICNa and SLC were 0.80±0.07 and 0.61±0.10, respectively. We performed a genome screen using a maximum-likelihood based, variance components linkage analysis program (SOLAR) and obtained evidence that a possible quantitative trait locus (QTL) for SLC is located on the baboon homologue of human chromosome 4 between D4S2456 and D4S2365 with a maximum multipoint lod-score = 8.76 (p -10 ) at D4S1645. This QTL accounts for approximately 2/3 of the additive genetic variation in SLC in baboons. We found no evidence for linkage with ICNa. Thus, we have evidence that a gene located on human chromosome 4 (baboon chromosome 5) affects cell sodium transport in baboons. No strong candidate genes have yet been identified in this region. However, future studies to identify and characterize this gene may elucidate fundamental processes that contribute to the development of hypertension.

Foraging for Survival: Yearling Baboons in Africa (review)

Foraging for Survival: Yearling Baboons in Africa (review)

No Baboon is an Average Baboon

No Baboon is an Average Baboon

Myoclonia in Papio papio: are they all "epileptic"?

Myoclonia in Papio papio: are they all "epileptic"?

Guare's FOUR BABOONS ADORING THE SUN

Guare's FOUR BABOONS ADORING THE SUN

Baboon Tales. Narrated by Glenn Close

Baboon Tales. Narrated by Glenn Close

Foraging for Survival: Yearling Baboons in Africa

Foraging for Survival: Yearling Baboons in Africa

Foraging for survival: Yearling baboons in Africa

Foraging for survival: Yearling baboons in Africa

Foraging for Survival: Yearling Baboons in Africa. Stuart A. Altmann

<i>Foraging for Survival: Yearling Baboons in Africa</i>. Stuart A. Altmann