Mineral Oil Aspiration Research Articles

PRESUMED MINERAL OIL ASPIRATION AND CAVITARY LUNG LESIONS IN A DOG

This report describes an unusual occurrence of cavitary lung lesions which may have been secondary to aspiration of mineral oil and illustrates a possible danger of aspiration pneumonia associated with administration of lipids. Lipid pneumonia should be considered in the differential diagnosis for pulmonary disease with radiographic signs ranging from increased interstitial opacity to nodules or cavitary lung lesions. This report also emphasizes the benefits of serial radiography to detect changing pathology and the occurrence of complications such as pneumothorax.

Lipoid pneumonia secondary to baby oil aspiration

Baby oil is a common household product that is frequently used when there are infants or toddlers in the house. However, it is often overlooked as a potential source of danger to these youngsters. In 1983, 36,700 cases of ingestion were reported to the poisoning surveillance and epidemiology branch of the Food and Drug Administration. Topical preparations used in the care of infants accounted for 480 of the cases. Ten percent of these required hospitalization. In 36 cases, the product ingested was baby oil. This figure does not include baby lotions and other skin products with a mineral oil base. Aspiration of mineral oil, the main component of baby oil, has been described as a cause of lipoid pneumonia and oleomas. However, there is very little information in the modern literature concerning acute lipoid pneumonitis in children. We herein present a patient with lipoid pneumonia caused by aspirated baby oil, who followed a severe clinical course. The paucity of information regarding this subject points to the need for increased public and physician awareness of the problem and for their direct participation in the prevention of this potentially fatal condition.

Lipoid pneumonia secondary to baby oil aspiration

Baby oil is a common household product that is frequently used when there are infants or toddlers in the house. However, it is often overlooked as a potential source of danger to these youngsters. In 1983, 36, 700 cases of ingestion were reported to the poisoning surveillance and epidemiology branch of the Food and Drug Administration. Topical preparations used in the care of infants accounted for 480 of the cases. Ten percent of these required hospitalization. In 36 cases, the product ingested was baby oil. This figure does not include baby lotions and other skin products with a mineral oil base. Aspiration of mineral oil, the main component of baby oil, has been described as a cause of lipoid pneumonia and oleomas. However, there is very little information in the modern literature concerning acute lipoid pneumonitis in children. We herein present a patient with lipoid pneumonia caused by aspirated baby oil, who followed a severe clinical course. The paucity of information regarding this subject points to the need for increased public and physician awareness of the problem and for their direct participation in the prevention of this potentially fatal condition.

Honeycomb Lung (Interstitial Fibrosis)

<h3>Common</h3> [Cystic bronchiectasis] Histiocytosis X (especially eosinophilic granuloma) Idiopathic interstitial fibrosis (Hamman-Rich ) Pneumoconiosis Sarcoidosis <h3>Uncommon</h3> Amyloidosis Chemical inhalation (late stage) Desquamative interstitial pneumonitis (DIP) Drug sensitivity, chronic or recurrent (eg, nitrofurantoin, hexamethonium, busulfan) Gaucher disease Lipoid pneumonia (eg, mineral oil aspiration) [Lymphangiomyomatosis] Oxygen toxicity [Mucoviscidosis] Neurofibromatosis Rheumatoid lung Scleroderma; dermatomyositis Sensitivity pneumonitis, recurrent (eg, farmer's lung, bagassosis) Tuberous sclerosis (Entities in brackets can be confused roentgenographically with true honeycomb lung. ) <h3>Diagnosis</h3> Eosinophilic granuloma. <h3>COMMENTS</h3> In analyzing this Gamut, a number of the possibilities offered, such as chemical inhalation, rheumatoid lung, and scleroderma are excluded by the lack of specific clinical information. Of the common causes, cystic bronchiectasis tends to show fluid in the cysts and to expand on inspiration; pneumoconiotics have a history of dust exposure. Therefore, idiopathic interstitial fibrosis (the

Mineral Oil Pneumonia

Excerpt Mineral oil pneumonia may be defined as the inflammatory, granulomatous, and fibrotic reaction of the lung to the aspiration of mineral oil. It is to be differentiated from endogenous lipoi...

Lipoid Pneumonia Associated with Paraesophageal Hernia: Angiocardiographic Study of a Case

An asymptomatic adult with bilateral lower lung field infiltrations went 13 years before the history of nightly ingestion of mineral oil was elicited. An unsuspected paraesophageal hernia was also found and may have contributed to mineral oil aspiration and lipoid pneumonia. Bronchography showed obstruction of the bronchi leading to the involved segments. Angiocardiography revealed avascularity of the lung in the regions of the lipoid areas, probably because the pulmonary circulation was destroyed by fibrotic changes early in the disease. A year after cessation of mineral oil ingestion the patient was asymptomatic; no change in the chest roentgenogram could be detected. Surgical repair of the paraesophageal hernia did not seem warranted because of the asymptomatic state.

Pulmonary Hazard of the Ingestion of Mineral Oil in the Apparently Healthy Adult

PERIODIC reports of harm resulting from the ingestion or aspiration of mineral oil have appeared in the medical literature and even in the lay press. Recently, it has been shown that the taking of liquid petrolatum with food prevents substantial amounts of food carotene, a precursor of vitamin A, from entering the body.1 This fact and some corollaries thereto have been established to the satisfaction of authors of the latest books on nutrition and vitamin therapy and have provoked editorial comment.2 More than two decades have elapsed since it was demonstrated that serious harm can be done to the lungs . . .