Previous studies showed that disturbed in the shear rate (SR) patterns, characterized by high retrograde and oscillatory SR on conduit artery, are associated with pro-atherogenic phenotype. This phenomenon seems to be caused by increased sympathetic nervous activity and reduced bioavailability of nitric oxide. Considering the anabolic androgenic steroids (AAS) abuse induces autonomic dysfunction, it is possible to speculate that AAS users present alterations in the SR on conduit artery. PURPOSE: The aim of this study was to evaluate the shear rate patterns in young men under AAS abuse. METHODS: 18 volunteers were divided into 2 groups: self-reported Anabolic Androgenic Steroid Users (AASU, n=10), Anabolic Androgenic Steroid Non-Users (AASNU, n=8), both group were bodybuilder (strength exercise training). The patients were submitted to evaluate SR patterns and flow-mediated dilation (FMD) both in the brachial artery. Furthermore, carotid artery intima-media thickness (IMT) was evaluated. All vascular variables were obtained by Doppler ultrasound and the images were analyzed analyzed by brachial analyzer. RESULTS: Age was similar among AASU and AASNU (27±1 vs. 27±1 years, p=0.87). Anterograde SR (118.0±11.0 s-1 vs. 117.0±17.0 s-1, p=0.9) and mean SR (78.0± 12.0 s-1 vs. 98.0±21.0 s-1, p=0.3), were similar between groups. AASU showed higher retrograde SR (-42.0±6.0 s-1 vs. -19.0±5.0 s-1, p=0.01) and oscillatory SR (0.30±0.03 uu vs. 0.16±0.04 uu, p=0.03) compared to AASNU. AASU showed lower FMD compared to AASNU (7.2 ± 0.7% vs. 9.6 ± 0.7%, p=0.04). In addition, AASU showed higher carotid artery IMT compared to AASNU (0.62±0.02 mm vs. 0.49±0.02, p=0.005). CONCLUSION: Our results suggest that AAS abuse induces disturbed in the SR patterns in the brachial artery and endothelial dysfunction. These alterations could increase the risk of developing atherosclerosis in young men under AAS abuse.