Accessory Renal Vessels Research Articles

Colour Doppler ultrasound in renal artery stenosis: intrarenal waveform analysis

Renal artery stenosis (RAS) is the commonest secondary cause of hypertension and may result in renal ischaemia with resultant renal failure. Recent studies hve suggested that colour Doppler ultrasound, with spectral analysis of the intrarenal waveforms, can identify those patients with a significant RAS. A prospective study was performed in which colour Doppler ultrasound was compared with angiography in 73 patients (143 kidneys) presenting for renal angiography. Colour Doppler ultrasound was unsuccessful in 16% of kidneys due to a combination of technical failures and small kidney size. Accessory renal vessels were present in 14% of kidneys on angiography but none was detected by ultrasound. Of the 120 kidneys that had both examinations, no significant difference in intrarenal pulsatility or resistive index was noted between the angiographically stenosed and normal arteries. There were significant differences for intrarenal peak and end diastolic velocities, and acceleration time and index. Of these measurements, acceleration time was the best indicator of RAS. The probability of detecting a high grade RAS in an individual patient did not reach 90% until the acceleration time was prolonged to more than 0.12 s. Intrarenal colour Doppler ultrasound is not a general screening test for RAS and it should be reserved for selected patient groups where the incidence of disease is high. Patients with prolonged acceleration times of more than 0.12 s have a high likelihood of at least 70% RAS and should proceed directly to angiography.

Ureteropelvic junction obstruction caused by accessory renal vessels in association with preureteral vena cava and vena caval duplication

An apparent ureteropelvic junction obstruction on the right side may herald an array of anomalous conditions for which the surgeon should be prepared. A case is presented in which ureteropelvic junction obstruction caused by accessory renal vessels was found in association with a preureteral vena cava and a vena caval duplication on the right side. The embryology of caval anomalies associated with preureteric vena cava is reviewed.

Catheter Angiography for Abdominal Aortic Aneurysms—Is It Still Necessary?

From 1985 to 1988, 205 patients were admitted to the authors' department for abdominal aortic aneurysm; 152 of them underwent preoperative conven tional catheter angiography (CA). These arteriograms were evaluated retrospec tively. Mean inner aneurysm diameter was 7.72 cm, which correlated well with sonographic findings. In 16.4% the diameter exceeded 10 cm. Aneurysms of the common iliac were present in 59.9%. In 21.0% we found stenoses of the common iliac vessels and in 16.4% stenoses of the external iliac arteries. Renal artery stenosis of more than 50% was diagnosed in 15.8% . In 6.6% accessory renal vessels were visualized, which had to be spared during aneurysm repair. In 9.2 % the inferior mesenteric artery was occluded, whereas all other visceral vessels were patent without pathologic lesions. Although newer noninvasive techniques are available today for vascular imaging, CA still is the "golden standard" for evaluating abdominal aortic aneurysms. It is the only method providing all the information a vascular surgeon needs for optimal aneurysm repair.

Abdominal Pain, Intermittent Hydronephrosis, and Accessory Renal Vessels

THE DIFFERENTIAL diagnosis of the patient with vague upper abdominal pain apparently arising in the right upper quadrant includes the standard considerations such as cholecystitis, ulcer disease, and functional bowel distress. This last diagnosis was made in several of the patients whose cases are reported here and who later proved to have intermittent hydronephrosis caused by accessory renal vessels. Unless specific questions are put regarding the periodicity of the pain and its relationship to maintained positions of the body and/or the ingestion of large quantities of fluid, the correct diagnosis may not be suspected. When the diagnosis is considered and an excretory urogram is performed in the absence of the pain, the hydronephrosis may not be demonstrated.^1,2The necessity of performing the urogram during an attack of pain is emphasized herein. The data on nine young adults with this syndrome observed during the past ten years at the University

Abdominal pain, intermittent hydronephrosis, and accessory renal vessels

Abdominal pain, intermittent hydronephrosis, and accessory renal vessels

The Diagnosis of Hydronephrosis Caused by Accessory Renal Vessels

No AccessJournal of Urology1 Dec 1944The Diagnosis of Hydronephrosis Caused by Accessory Renal Vessels Earl C. Lowry, Joseph C. Hayward, and Donald E. Beard Earl C. LowryEarl C. Lowry More articles by this author , Joseph C. HaywardJoseph C. Hayward More articles by this author , and Donald E. BeardDonald E. Beard More articles by this author View All Author Informationhttps://doi.org/10.1016/S0022-5347(17)70293-5AboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissionsReprints ShareFacebookLinked InTwitterEmail © 1944 by The American Urological Association Education and Research, Inc.FiguresReferencesRelatedDetailsCited ByMalament M and Majnarich G (2018) Giant Hydronephrosis in Crossed Renal Ectopia without FusionJournal of Urology, VOL. 83, NO. 5, (542-545), Online publication date: 1-May-1960.Hoffman H (2018) Massive HydronephrosisJournal of Urology, VOL. 59, NO. 5, (784-794), Online publication date: 1-May-1948.Zimet R and Kappel L (2018) Giant Hydronephrosis as a Cause of Pyonephrosis in the Opposite Kidney: Report of a CaseJournal of Urology, VOL. 56, NO. 5, (515-519), Online publication date: 1-Nov-1946. Volume 52Issue 6December 1944Page: 492-496 Advertisement Copyright & Permissions© 1944 by The American Urological Association Education and Research, Inc.MetricsAuthor Information Earl C. Lowry More articles by this author Joseph C. Hayward More articles by this author Donald E. Beard More articles by this author Expand All Advertisement Loading ...