Abstract
Abstract Glioma is the most common and aggressive primary brain tumor in clinic. Due to its high heterogeneity and not-fully understood pathogenesis, the progress of clinical treatment of glioma is slow. Current studies have demonstrated that Ets-1 (E26 tansformation-specific-1) is a proto-oncogene. Our immunohistochemical analysis indicated that the expression level of Ets-1 in glioma tissues was significantly higher than that in normal brain tissues ( P P
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