Abstract
A female 82-year-old Caucasian patient with a history of paroxysmal atrial fibrillation (AF) for several years, with palpitation crises of varying duration between a few minutes and several hours, without clinical control, using beta-blockers and diltiazem. The patient presented complaints of fatigue on moderate efforts, without precordial pain or syncope, with progressive asthenia and indisposition. She informed three previous hospitalizations for chemical cardioversion of AF.
Highlights
The patient had a history of systemic arterial hypertension, hypothyroidism, and atherosclerotic heart disease, and underwent coronary angioplasty in 2014 with the placement of a nonpharmacological stent, without a prior heart attack
This decision was based both on the inefficiency of the drug in controlling atrial fibrillation (AF) crises and on the interpretation that propafenone could have some role in the electrocardiographic pattern of SBr
Brugada syndrome (SBr) is a clinical entity characterized by the presence of a pattern of right bundle-branch conduction disorder, ST-segment elevation in right precordial derivations, risk of severe ventricular tachyarrhythmias and sudden cardiac death in patients with a structurally normal heart[1,2]
Summary
The patient had a history of systemic arterial hypertension, hypothyroidism, and atherosclerotic heart disease, and underwent coronary angioplasty in 2014 with the placement of a nonpharmacological stent, without a prior heart attack. Her electrocardiogram (Figure 1) showed the presence of sinus rhythm, mild interatrial conduction disorder (P wave duration of 125 ms), AV interval at the upper limit of normality (0.20 s), QRS with 125 ms duration and presence of isolated ventricular extrasystoles.
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