Abstract
We agree with Dr Ostadal that the current terminology used to describe the range of changes induced by the reperfusion of myocardium which has undergone prolonged ischaemia can be both confusing and inadequate. While the spectrum of changes could be described under the umbrella term of ischaemia/reperfusion injury, this term itself is too broad as the pathophysiology of the resultant ischaemia/reperfusion substrate will depend on the severity, transmural extent, and duration of the pre-existing ischaemia, as well as on the type of reperfusion therapy used, the characteristics of the reconstituted flow (driving pressure), and the degree of …
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