Abstract
What happened to a thrombus during apical ballooning syndrome: a case report
Highlights
Impaired left ventricle (LV) function, increased LV volume, apical aneurysm or endocardial injury might promote left ventricle thrombosis (LVT) in both anterior myocardial infarction (AMI) and apical ballooning syn
Evidence on apical LVT in ABS is mainly based on single reports and series of clinical cases, it is known that LVT has peculiar characteristics in ABS or AMI
Though ABS is often associated with severe acute LV dysfunction, LVT was documented in 5.3% of cases and cardioembolic complications occurred in less than 1% in different studies [4, 5]
Summary
Impaired left ventricle (LV) function, increased LV volume, apical aneurysm or endocardial injury might promote left ventricle thrombosis (LVT) in both anterior myocardial infarction (AMI) and apical ballooning syn-. Impaired left ventricle (LV) function, increased LV volume, apical aneurysm or endocardial injury might promote left ventricle thrombosis (LVT) in both anterior myocardial infarction (AMI) and apical ballooning syndrome (ABS). A routine electrocardiogram showed anterior-lead ST-segment elevation; transthoracic echocardiography (TTE) revealed apical ballooning, impaired LV ejection fraction (40%) and a stratified LVT
Sign-in/Register to view highlights & summary Sign-in/Register to access full text options 
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
