Abstract
It is difficult to dissect out the contribution of ventilation versus other stimuli in the progression of lung injury in ARDS. Earlier this century, hyaline membranes and the histologic findings currently associated with ARDS were rarely found1, 2. Following the widespread introduction of positive pressure ventilation into clinical practice, the term “respirator lung” was coined to describe findings of dense cellular infiltrates, pulmonary edema, and hyaline membranes upon post-mortem examination of patients subjected to respiratory support. Subsequent studies have characterized a number of histologic findings typical of lung injury in ventilated patients.
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