Abstract
Background and Aims: Progression of atherosclerosis is associated with pathological remodelling of native vascular smooth muscle cells (VSMC) driven by platelet-derived growth factor (PDGF) signalling and subsequent activation of the calcium ion channel, Orai1. The quest now is for strategies to specifically target Orai1 channels and for comprehensive analysis of the impact of Orai1 dysfunction on vascular physiology and pathology.
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