Abstract
Environmental conditions are critical in the susceptibility and severity of autoimmune disorders. Multiple Sclerosis (MS) is characterized by a positive latitude gradient (the further away from the equator, the higher the disease incidence). Here, we investigated the mechanisms underlying the beneficial effects of immune regulatory processes induced in the skin by moderate ultraviolet B (UVB) radiation on central nervous system (CNS) autoimmunity. Effects of UVB light were analyzed in a murine model of CNS autoimmunity (experimental autoimmune encephalomyelitis (EAE)).
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