Abstract
Objectives: The cardioprotective effects of macrophage migration inhibitory factor (MIF) in myocardial ischemia and reperfusion (I/R) seem to be further increased by application of the soluble form of MIF receptor CD74 ectodomain (sCD74). Therefore, we aimed to assess the underlying mechanisms of enhanced myocardial protective effects of MIF/sCD74 complex formation by stimulating cardiac cells with MIF alone or in complex with sCD74.
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