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Abstract
We read with great interest the paper by Shao et al. highlighting the possible shared mechanism underlying McConnell's phenomenon and reverse Takotsubo cardiomyopathy (TTC) [1]. The authors brilliantly relate ventricular wall overstretching induced by abrupt increase in afterload and transient ventricular dyskinesis observable in pulmonary embolism (McConnell's sign [2]) with basal dyskinesis characterizing reverse TTC [3]. The authors also remarked that myocardial ischemia possibly induced by increased local wall stress may further precipitate myocardial stunning observable in both conditions, usually featured by local dyskinesis and generally related to catecholamine toxicity in TTC.
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