Abstract
Myasthenia gravis (MG) primarily affects skeletal muscle through the production of autoantibodies against the nicotinic acetylcholine receptor in the motor endplates; in addition, heart muscle is an autoimmune target. We describe 2 illustrative cases of myasthenia gravis (MG)-related cardiomyopathy. The first case is a 63-year-old man treated for the decreased left ventricular systolic function and the positive results of autoantibody against striational proteins. The self-discontinuation of immunosuppressive agents was suggested to result in acute decompensated heart failure with serum levels of cardiac troponin I of 0.461 ng/mL and death.
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