Treatment of universal hyperpathy and pain threshold control

Abstract

This article presents a disease fundamentally characterized by an alteration of the pain threshold. In this disease, the pain threshold can change and, in general, is lower, so pain is perceived before. The cause of the decrease is a continuous pressure stimulus of intensity above or close to the threshold that persists over time. The duration to reduce the threshold will depend on the degree of intensity: It will be longer if it is of lower intensity. In the absence of medication, the decrease in threshold is characterized by a sensation of electrical pain followed by allodynia, hyperalgesia, and dysesthesia in the affected area. The level of pain tolerance in that area drops, so the patient cannot support that area. The disease becomes completely disabling. The patient was diagnosed with universal hyperpathy; in fact, the decrease in the threshold can affect both somatic and visceral pain. Although the disease itself would have a minimum level of incidence worldwide, since the probability of survival of individuals is small, the content presented here is useful for the treatment of hyperpathy and the control of the pain threshold. Carbamazepine and topiramate turn out to be two medications that allow modulation of the threshold. Performing aerobic exercise is also essential. Furthermore, it is necessary to control the stimuli of the afferent pathways at all times, both in duration and intensity.