Abstract
There has been a longstanding controversy about whether high blood pressure should be treated in the setting of acute stroke.1,2 Normally, cerebral blood flow is maintained through a wide range of systemic mean arterial blood pressure, from &50 to 150 mm Hg.3,4 In the setting of cerebral ischemia (and probably also in the zone of injury around intracerebral hemorrhages), the ischemic zone partially loses autoregulation, so cerebral blood flow in that region becomes dependent on perfusion pressure.5 Many experts, therefore, recommended that blood pressure elevation, which is common in the setting of acute stroke, not be treated for fear of exacerbating stroke by reducing perfusion pressure and thereby reducing flow in the compromised but viable ischemic penumbra. Because swelling in the region …
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