Abstract
It is already known that myocardial diastolic dysfunction develops already at early stages of I type diabetes mellitus. Current investigations represent the importance of oxidative stress in the development of complications of diabetes mellitus. The purpose of our work was to show the role of oxidative stress in progress of disturbances of myocardial relaxation function in rats with streptozotocin-induced diabetes and to explore possible mechanisms of heart protection. The essential increase of left ventricular diastolic stiffness (considered as ΔEDP/ΔV) is represented with Langendorff isolated heart of rats that had experimental diabetes.
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