Abstract
The rupture of advanced atheromatous plaques initiates a significant number of cases of coronary artery thrombosis and subsequent myocardial infarction. Hypothesis : The range of outcomes following plaque rupture is determined to a large extent by the interaction between the turbulent arterial stream and the visco-elastic properties of the extruded atheromatous material. Thrombosis is a secondary event. Research into the visco-elastic behaviour of atheromatous material may predict predisposition to myocardial infarction.
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