Abstract
In some forms of obesity, the accumulation of fat in “bad places”, that is, somewhere else than in adipocytes, seems to be responsible...
Highlights
In some forms of obesity, the accumulation of fat in “bad places”, that is, somewhere else than in adipocytes, seems to be responsible for diabetes associated with impaired insulin signals
Many epidemiological studies indicate a relationship between obesity and diabetes and to better understand this relationship, we found it necessary to study the links between serum levels of various variables including insulin, vitamin E, and some cations involved in a major metabolic process incriminated in the pathophysiology of these two diseases: oxidative stress (we study magnesium (Mg), zinc (Zn) and calcium (Ca)), in obese diabetic type 2 and obese non diabetics
The Body Mass Index (BMI) is between 30 - 47Kg / m2 and on average it is 38. 5Kg / m2. They are all non-smokers and have no other diseases associated with their obesity. These two groups of diabetic obese and nondiabetic obese are compared to 30 control subjects matched by age and sex with a mean age of 52 years and an average BMI of 22.4Kg / m2
Summary
In some forms of obesity, the accumulation of fat in “bad places”, that is, somewhere else than in adipocytes, seems to be responsible for diabetes associated with impaired insulin signals. Free radicals are harmful to the body, their frequent accumulation in the cells of obese subjects generally leads to the disruption of the role of insulin and the development of a state of insulin resistance preparing for the installation of true non-insulin-dependent diabetes. For this reason, it seems important to take this oxidative stress in obese and diabetic for better treatment. Many epidemiological studies indicate a relationship between obesity and diabetes and to better understand this relationship, we found it necessary to study the links between serum levels of various variables including insulin, vitamin E, and some cations involved in a major metabolic process incriminated in the pathophysiology of these two diseases: oxidative stress (we study magnesium (Mg), zinc (Zn) and calcium (Ca)), in obese diabetic type 2 and obese non diabetics
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