Abstract
Isolated perfused hearts of guinea-pigs continuously release a prostaglandin-like substance into the effluent. Polyunsaturated fatty acids, linoleic acid and arachidonic acid > linolenic acid, induced an increase in PG-efflux. Oleic acid was without effect. The results suggest that PG-formation is involved in the increase of contractile force and coronary flow after administration of linoleic, linolenic and arachidonic acids.
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