Abstract
The extra endogenous protein metabolism occurring in phlorizin and alloxan diabetes did not significantly change the liver arginase activities of normal or castrated adult male rats. Moreover, no correlation between the degree or duration of the diabetes and the enzyme activities of the liver could be detected. Tremendous increases, 400–600%, in protein ingestion, however, stimulated identical increases in the arginase activities of the livers of diabetic and nondiabetic rats. Smaller increases, 100–200%, produced increases in arginase activity concomitantly with the increase in liver weight. Testosterone propionate did not affect these enzymic changes. The arginase activity of the kidney was not changed except in those rats that had considerable sclerosis of the tubules after prolonged and severe alloxan diabetes. The ability of testosterone propionate to increase the arginase activity of the kidney was not impaired by the diabetes.
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