Abstract

SummaryCleft lip and palate (CLP) is a risk indicator to middle ear alterations, which may damage the development of auditory abilities such as attention that is essential to learn new skills, oral and written communication. Studies on attention process with CLP population are recent and poorly explored in the specific literature. Thus, this study aims to contribute with new subsidies in the field as it investigates the performance of children with CLP in Sustained Auditory Attention Ability Test (SAAAT).Material and MethodComparison of SAAAT performance between children with CLP and children without it. Prospective study.ResultsANOVA was used as variance analysis model with two factors to study the variables such as group and gender. The CLP group showed an average of 2.5 units higher than the control group. This difference is between 0.7 and 4.4 with 95% certain.Conclusionchildren with cleft lip and palate had poorer performance on SAAAT when compared to those without such craniofacial anomaly, considering attention reduction only.

Highlights

  • Congenital cleft lip and palate (CLP) develop in the face during the embryonic and initial fetal stages

  • This is due to lack of palate muscle fusion, which supports the theory that middle ear hypoventilation may be a cause of otitis media with effusion (OME)

  • The Sustained Auditory Attention Ability Test (SAAAT) was applied to all participants in this study

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Summary

Introduction

Congenital cleft lip and palate (CLP) develop in the face during the embryonic and initial fetal stages. It is known that otitis media with effusion (OME) is almost universally present in this population because of auditory tubal dysfunction This is due to lack of palate muscle fusion, which supports the theory that middle ear hypoventilation may be a cause of OME. OME is a special form of otitis media; it installs itself silently, and is characterized by the accumulation of serous or a glue-like mucous liquid in the middle ear (glue ear). Today, this disease is one of the most common causes of often bilateral hypoacusis in children up to age 10 years.[2]

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