Abstract
Sudden cardiac arrest resulting from myocardial infarction (MI) is most often due to ventricular tachycardia (VT). In the infarcted heart, VT has been shown to originate at the periphery of the scar1. Such tissue is often termed the infarct border zone (BZ) and is comprised of a heterogeneous admixture of fibrosis and surviving cardiomyocytes2. Extended regions of BZ tissue that penetrate through non-conducting necrotic core scar can give rise to anatomical isthmuses which provide slow conducting re-entrant pathways that can help sustain VT circuits3.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
