Abstract
Aphasia, a language deficit caused by an acquired cerebral lesion was initially believed to be triggered exclusively by cortical damage. However, recent findings have confirmed that aphasia can also arise from subcortical lesions. This paper presents a case of a 53-year-old man with acquired language impairment associated with a lesion in the left lenticular nucleus caused by a hemorrhagic cerebrovascular event. The patient exhibited clinical and imaging characteristics consistent with subcortical aphasia of the thalamic and striato-capsular type. Subcortical structures are increasingly recognized for their involvement in language processing, alongside the cerebral cortex. Despite current acceptance of subcortical aphasia, there are limited studies on this type of aphasia and its interlinguistic variations. Consequently, extensive research is warranted to gain a better understanding of it as a clinical entity.
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