Abstract
Several investigators have observed, under conditions of little or no gas exchange, that large differences in PCO2 can occur between mixed venous blood and alveolar gas (Jones et al: 1969; Gurtner et al: 1969; Dennison et al: 1969, Guyatt et al, in press). Gurtner et al (1969) have found that ΔPCO2 was related to both [H+] and [HCO3-] activity by the mixed venous blood. They developed a model explaining their results which involves a coupling of bulk flow, diffusion and chemical reaction near a negatively charged capillary wall.
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