Abstract
The Switch/Sucrose Non-Fermentable (SWI/SNF) protein complex functions as a tumour suppressor through chromatin remodelling. Mutations in genes that encode complex subunits have been identified in a rare subset of aggressive poorly differentiated malignancies across anatomical sites, including thorax, gastrointestinal, genitourinary and sinonasal tracts.1,2 The mutations are independent predictors of poor outcome. Cases presenting as isolated lymphadenopathy are uncommon.3
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