Abstract
The present investigation reports on the accumulation and damage caused by copper in the liver and kidney of albino rats. Simultaneous treatment with copper and molybdenum shows that no traces of copper are retained in these tissues. Thus these observations lead to the conclusion that copper is ineffective in the presence of molybdenum exhibiting physiological antagonism. Several hypothesises have been considered for their antagonistic behaviour. Present authors suggest that molybdenum may act on copper as a chelating agent binding with it and forming either cupric molybdate or copper thiomolybdate which is ultimately excreted. Intake of molybdenum with the diet has also been suggested for the occupational victims of copper.
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