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PDF Availablehttps://doi.org/10.1128/iai.69.1.589-592.2001
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Cite Journal: Infection and immunity |  Publication Date: Jan 1, 2001 |
 Citations: 28 |
An interleukin-4 (IL-4)-dependent Th2 response allows wild-type mice to survive infection with the parasite Schistosoma mansoni. In the absence of IL-4, infected mice mount a Th1-like proinflammatory response, develop severe disease, and succumb. Neither the Th1 response nor morbidity is IL-12 dependent in this system.
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