Abstract
Aberrant gamma-aminobutyric acid (GABA) system, the major inhibitory neurotransmitter in the CNS, is highly implicated in autism spectrum disorder (ASD). Development of the GABAergic neurons in human has only been recently examined, and the results unexpectedly show that GABA neurons are overproduced in ASD (Mariani et al 2015). It is essentially unknown as to how GABAergic transmission is perturbed in developing ASD circuitry.
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