Abstract
The initiating events happening during store-operated calcium entry are not completely understood despite the tremendous importance of this process in immune responses. Upon endoplasmic reticulum (ER) calcium depletion, STIM1 and Orai1 form calcium release-activated calcium (CRAC) channels to allow calcium influx from the extracellular milieu. S-acylation is a reversible post-translational modification of cysteine residues that affects protein structure, stability, and targeting to subdomains in the plasma membrane.
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