Abstract
Acute Kidney Injury (AKI) remarkblely affect public health and need higher healthcare costs, which is life-threatening and characterized of acute decline with function. Arachidonic Acid (AA), the precursor for synthesis of various biologically active molecules and be involved in regulation of homeostasis and metabolism. Recent studies have shown that the AA cytochrome P450 (CYP450) pathway contribute to the development of AKI by EETs (the product of catalyzing epoxidation reactions), 20-HETE (the product of omega-hydroxylation reactions) and maybe potent key for the prevention and treatment of AKI. This review summarizes recent advances in AA metabolites in AKI.
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