Abstract
Experiments on rats showed that chronic administration of the β-adrenoblocker propranolol in doses blocking glycogenolytic and lipolytic effects of exogenous adrenalin, does not prevent the fall in the glycogen level in the liver and myocardium and the rise in the serum free fatty acid level and in the lipolytic activity of adipose tissue in vitro arising under the influence of large doses of thyroxine.
Published Version
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