Abstract
Background and Aims : Endothelial dysfunction induced mostly by oxidized sterols and microbial components is one of the main causes of atherosclerotic lesion formation. Understanding the mechanisms involved in the process of development and progression of proatherogenic changes may help to find different ways to modulate the course of coronary heart disease (CHD). Our study aimed on analyzing the role of various sterols in the process of vessels regeneration, also in the environment of bacterial components.
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