Abstract
Gout has increasingly been linked to the development of numerous cardiovascular diseases and cardiac events. The inflammatory processes underlying the pathophysiology of many cardiovascular diseases are similar or identical to those responsible for gout. This article will discuss the pathogenesis of gout, the inflammatory processes that drive it, and the similar process seen in numerous cardiovascular diseases. By viewing gout as a symptom of systemically high levels of inflammation and a possible harbinger of cardiovascular diseases, we can decrease the risk of development of associated cardiovascular conditions and adverse cardiac events. This review will also investigate possible inflammatory markers that can act as drug targets to decrease inflammation, ameliorate symptoms, and reduce the risk of heart disease.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callSimilar Papers
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
