Abstract
Abstract Damage to striate cortex (V1) in primates does not result in absolute blindness, but instead spares some rudimentary visual functions such as the ability to detect and localize visual stimuli in the affected visual field. In both humans and monkeys, early brain injury is typically associated with greater sparing or recovery of function, although this is not an invariable rule. We compared the visual function in monkeys with lesions of V1 sustained either in infancy or adulthood and found greater residual vision after the V1 lesions made in infancy. We describe the potential of these observations to explain the variability in the extent of residual visual function present in human subjects with damage to V1 and consider the possible mechanisms of the greater residual function after early V1 damage in primates. We also discuss the conspicuous similarities between the residual vision of monkeys with adult V1 lesions and human blindsight.
Published Version
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