Abstract

Abstract Background Assisted ventilation represents high benefits for patients that cańt breathe normally or had suffered lung damage. The problem begins when there is an excessive assisted ventilation, that could lead to partial pression of the oxygen that cause hyperoxia and the result is oxygen poisoning [Hyperoxemia]. Reports have shown that cells, connective tissue, and organs can be affected by the hyperoxia. Our research consists in identifying hyperoxemia prevalence in Intensive Care Unit [ICU] at hospitals, so they can develop a better oxygen treatment. Methods The method we use in this investigation were: Nonexperimental, quantitative, retrospective, and transversal research, with the variable data PAO2, PCO2, SO2, age and gender. The subjects were distributed in normoxia and hyperoxemia. The population sample was of 19 patients from the Hospital A, B, and C in the south of Tamaulipas, Mexico, during the August-September 2019 period. The analytic technique used was the statistical average, which was of 137.9% in ICU. Results From 19 patients [100%], 15 patients with hyperoxemia [79%] and 4 patients with normoxemia [21.05%]. There was a difference between hyperoxemia: 10 male patients [52.63%] and 5 female patients [26.31%]. In the ICU, oxygen average saturation was 96.7%, and partial pressure of oxygen was 137.9% Conclusions We recommend performing a blood gas test in each patient with assisted ventilation, this with the purpose of regulate the administration of the oxygen inspiratory rate [FiO2]. Develop a clinic practice manual to regulate the administration of the oxygen inspiratory rate [FiO2] with assisted ventilation patients, with specifications and parameters to use it according each patient's characteristics and noted the consequences of a bad management on this. From 100% of patients [19], 79% [15] had hyperoxemia, which denotes that it is missing health surveillance to look after patient's health in the future. Key messages There are no beneficial effects of hyperoxemia in critical patients clearly demonstrated in clinical trials. The use of elevated FiO2 can produce a direct toxic effect on lung cells, with phenomena of cell destruction and alteration of defense mechanisms.

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