Abstract
Background and Aims: The research into the pathophysiology of atherosclerosis has considerably increased our understanding of the complexity of the disease, but still many questions remain unanswered, and the burden of recurrent cardiovascular events remains unacceptable, despite optimal treatment with contemporary intervention and pharmacologic agents. Our previous findings that prenylcysteine oxidase 1 (PCYOX1) represents a novel lipoprotein-associated protein, and that lipoproteins can themselves generate the reactive oxygen species H2O2 through the action of PCYOX1, prompted us to investigate the biological role of this unique enzyme in the development of atherosclerosis.
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