Abstract
The development of ischemic brain damage is dramatically affected by the immune system, whose activation occurs immediately after the insult and may last for several days, involving a complex interplay between soluble and cellular mediators (Amantea et al., 2015). Accordingly, recent expression profiling studies have revealed that the majority of the genes modulated in the blood of stroke patients participate in the regulation of innate immune responses (Brooks et al., 2014). Moreover, in the clinical setting, serum levels of markers of acute inflammation correlate with the severity of ischemic brain damage and neurological deficit.
Full Text
Published Version
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call