Abstract
AbstractPirlindole, a tetracyclic drug, is known to have a reversible and short‐lived monoamineoxidase inhibitory activity. When administered after a supramaximal inhibitory dose of a classic monoamineoxidase inhibitor, pirlindole continues to antagonize reserpine‐induced ptosis and oxotremorine‐induced akinesia in mice. These results indicate that some central effects of pirlindole are partially independent of its monoamineoxidase inhibitor activity.
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