Abstract
In 1935 FOERSTER and ALTENBURGER demonstrated by electrocorticography that brain tumors do not show spontaneous electrical activity. They hypothetized that focal EEG changes are due to the perifocal edema (FOERSTER and ALTENBURGER, 1935; WALTER, 1936). Later investigations with rCBF demonstrated a correlation between focal EEG changes and the degree of diminished cerebral blood flow (BROCK etal., 1966; HADJIDIMOS etal., 1969). At the present time, despite a plethora of data, the effects of tissue acidosis, deranged autoregulation, hypoxia, and even paradoxical “luxuryperfusion” (hyperemia) cannot be simply rela-ted to electrocorticographic changes in the peritumoral areas (HESS, 1975). CT is the only clinical method which visualizes the size and density of a perifocal lesion in space-occupying cerebral tumors (AMBROSE, 1973). So it seems interesting to investigate and quantify an influence of perifocal edema on focal EEG changes.
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