Abstract
Serotonin (5-HT) containing enterochromaffin (EC) cells of the intestine detect chemical and mechanical stimuli in the lumen and respond by releasing 5-HT on to afferent nerve terminals. Our recent electrochemical studies have shown that the real-time release of 5-HT is increased during inflammation, but the molecular changes are unknown. Our aim was to characterise the molecular basis of increased 5-HT availability in a mouse model of colitis.
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