Abstract
Oxidative stress in aging.
Highlights
The Alzheimer’s disease (AD) is the outcome of a complex interaction among several factors which are not fully understood yet; it is clear that oxidative stress and inflammatory pathways are among these factors
It has been described that sublethal oxidative stress could induce premature senescence of human mesenchymal stem cells derived from endometrium
Hyperandrogenism-induced polycystic ovary (PCO) in rats is associated with ovarian aging-like phenotypes
Summary
The AD is the outcome of a complex interaction among several factors which are not fully understood yet; it is clear that oxidative stress and inflammatory pathways are among these factors. The comparative effects of biodynes, tocotrienol-rich fraction, and tocopherol in enhancing collagen synthesis and inhibiting collagen degradation in stressinduced premature senescence model of human diploid fibroblasts have been described. Skin aging can be intrinsic or extrinsic, mediated genetically or due to environmental exposures.
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