Abstract
Spinal cord stimulation (SCS) utilizing a differential target multiplexed programming demonstrated significant improvements in pain-like behavior (i.e., mechanical and thermal hypersensitivity) in an animal model of chronic neuropathic pain. It also modulated gene expression within pain-related biological processes toward the levels of naïve animals1,2,3. This work was translated successfully to the clinic in patients with chronic pain4. This study presents results using reduced energy (RE) derivatives of Differential Target Multiplexed SCS in the same animal model.
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