Normal apical myocardial perfusion in the rat model with Takotsubo syndrome: is subsequent microvascular dysfunction and hypoperfusion an epiphenomenon?

Abstract

The pathophysiology of Takotsubo syndrome (TTS) continues to be elusive, but important recent contributions, like the one published on line on 5 July 2013 in the Journal ,1 will pave the way for the eventual unravelling of the mysteries surrounding this illness. The authors work focused on the crux of the pathophysiological conundrum: whether myocardial hypoperfusion is the cause, or a consequence of TTS. They referred to the literature about the conflicting reports showing myocardial hypoperfusion (often associated …