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Open Accesshttps://doi.org/10.1016/j.cmet.2010.02.004
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Cite Journal: Cell Metabolism |  Publication Date: Mar 1, 2010 |
 Citations: 43 |  License type: publisher-specific-oa |
Affiliation: University of Oxford
The discovery that their neonatal diabetes is caused by gain-of-function mutations in the K(ATP) channel has enabled many patients to switch from insulin to oral sulphonylurea drugs. Here, I review molecular, physiological, and clinical features of this change in therapy.
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