Abstract
Introduction: Myasthenia gravis (MG) is a rare autoimmune disorder of the neuromuscular junction associated with post-synaptic skeletal muscle membrane protein autoantibodies against the nicotinic acetylcholine receptors (ACh-R), muscle-specific kinase (MuSK) or agrin and lipoprotein receptor-related protein 4 (LRP4). The disease pathomechanism is complex, with predisposing human leukocyte antigen genes, pro-inflammatory cytokine profile, sex hormones, pharmacological agents, and environmental triggers. Several case reports suggest SARS-CoV-2 infection and Covid-19 vaccines as a possible trigger of MG.
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