Abstract
Pancreatitis is a painful inflammatory condition arising from pancreatic injury. Gallstone disease and alcohol abuse are the best known causes of pancreatitis, although more recently, cigarette smoking has emerged as a prominent risk factor. A wealth of clinical evidence underscores the role smoking plays in the disease, although knowledge of how cigarette toxins initiate pathological cellular events in the pancreas remains unclear. This mini-review focuses on recent research and highlights how cigarette smoke and smoking compounds may affect cell signaling pathways to initiate pancreatitis responses.
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