Abstract
Involvement of Fibrinogen(Fg) and fibrin during various pathologies associated with neuroinflammatory diseases associated with memory reduction are well known. Elevated level of Fg, called hyperfibrinogenemia (HFg) (e.g. ≥ 4±0.1 mg/ml of plasma during inflammation vs.~2±0.1 mg/ml of normal plasma [1]), accompanies inflammatory diseases such as stroke [2,3], hypertension [1,4], diabetes [5] and Traumatic Brain Injury (TBI) [6-9]. Blood level of Fg increases during inflammation in general [10].
Highlights
Severe acute respiratory syndrome coronavirus 2 (SARSCoV-2) is a novel virus, initially discovered in the city of Wuhan, China [1]
SARS-CoV-2 causes coronavirus disease (COVID-19), which has led to an ongoing global pandemic
The mechanism of action for SARS-CoV-2 may relate to a specific tropism for respiratory tract mucosal cells through the Copyright © All rights are reserved by Maria Stavrou, Solomis Solomou
Summary
Severe acute respiratory syndrome coronavirus 2 (SARSCoV-2) is a novel virus, initially discovered in the city of Wuhan, China [1]. Despite belonging to the coronavirus family, which usually cause self- limiting upper respiratory tract infections, SARS-CoV-2 is often more virulent than most coronaviruses and may lead to severe respiratory disease [2]. The mechanism of action for SARS-CoV-2 may relate to a specific tropism for respiratory tract mucosal cells through the Copyright © All rights are reserved by Maria Stavrou, Solomis Solomou. The virus causes a wide spectrum of disease severity, with most patients suffering a mild self-limiting disease. Dry cough and constitutional symptoms (headache, fatigue, myalgia, arthralgia), progressing to respiratory disease of mild to moderate severity [2,4]. The SARS-CoV-2 virus causes a wide spectrum of disease severity. Dry cough, and constitutional symptoms, which may progress to respiratory disease. There may be neurological and psychiatric manifestations, involving both the central and peripheral nervous system
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